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The optimal management of CKD-MBD (Chronic Kidney Disease – Mineral and Bone Disorder) should be achieved without increasing the risk of metastatic calcification, including that of blood vessels. 6. References Andress DL. (2006). “Vitamin D in chronic kidney disease: a systemic role for selective vitamin D receptor activation”. ; 69(1): 33-43. , (2000). “Increased risk of hip fracture among patients with end-stage renal disease”. ; 58(1): 396-9. Alfrey AC, LeGendre GR, et al. (1976). “The dialysis encephalopathy syndrome.

Foley RN, Parfrey PS et al. (1998). “Clinical epidemiology of cardiovascular disease in chronic renal disease. Am J Kidney Dis; 32 (5 Suppl 3):S112-9. , Y. Hamada, et al. (2006). “The kidney and bone metabolism: Nephrologists' point of view”. J Bone Miner Metab. 24(6): 434-8. Ganesh SK, Stack AG et al. (2001). ;12(10): 2131-8. Gao P, and D'Amour P (2005). ; 51(1-2): 21-9. Giachelli CM. (2004). ;15(12): 295964. Hollis BW and Napoli JL. (1985). ; 31(11): 1815-9. Huan J, Olgaard K, et al. (2006). ” JASN;17(7):1923-30.

CKD according to the National Kidney Foundation Kidney Disease Outcomes Quality Initiative (NKF–K/DOQI) classification (National Kidney Foundation: K/DOQI). In more advanced stages of CKD, the blunted urinary excretion of phosphorus can no longer keep pace with the obligatory intestinal phosphate absorption, resulting in hyperphosphatemia. , 1998). , 2005). Moreover, it has been shown also that these derangements in mineral metabolism could occur as well during the early stages of CKD (Slatopolsky and Delmez, 1994).

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