By Christopher G. Fairburn
This special guide provides and integrates nearly all that's presently recognized approximately consuming issues and weight problems in a single authoritative, available, and eminently sensible quantity. From top overseas professionals, 112 concise chapters encapsulate the newest info on all pertinent themes, from organic, mental, and social methods linked to danger, to scientific equipment for evaluate and intervention. feedback for additional analyzing on the finish of every bankruptcy change prolonged references and improve the sensible worth and clarity of the amount.
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Extra resources for Eating Disorders and Obesity, Second Edition: A Comprehensive Handbook
We will need all the power of human genetics, experimental genetics, and molecular biology to unravel the genetic basis of the predisposition to obesity. FURTHER READING Allison, D. , Neale, M. , Kezis, M. , Alfonso, V. , & Heymsfield, S. B. (1996). Assortative mating for relative weight: Genetic implications. —A discussion on assortative mating based on old and new evidence. The implications for population estimates of heritability are also highlighted. Bouchard C. (1996). Genetic and energy balance interactions in humans.
2 Peripheral Physiological Determinants of Eating and Body Weight GERARD P. SMITH JAMES GIBBS The relative importance of the peripheral controls of eating and body weight has increased significantly in the past 5 years. This has been due to the realization that peripheral controls function at every meal, that so-called long-term controls of food intake act by modulating the potency of the peripheral controls, and the discovery of a new genetic obesity in rats that is due to spontaneous mutation of a mechanism of a peripheral control.
Kendler, we explored the question using a population-based sample of 591 monozygotic (MZ) female twin pairs and 432 dizygotic (DZ) female twin pairs from Virginia. Thinness was defined as a current BMI < 20 kg/m2 (lowest 10th percentile for age) and no history of either anorexia or bulimia nervosa. Twin studies allow one to partition causal variance into three sources—additive genetic effects, shared environmental effects (to which both members of a twin pair are exposed, and which increase similarity between twins), and unique environmental effects (that act to increase differences between twins and to which only one member of a twin pair is exposed).